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High efficiency CRISPR knock-in demonstrates that TCF1 is insufficient to reverse T cell exhaustion

Organism

Mus musculus

Samples

3 samples

Published

Jan 15, 2026

Platform

19057

Type

Expression profiling by high throughput sequencing; Other; Genome binding/occupancy profiling by high throughput sequencing

Entry Type

GSE

Summary

In this study we investigated the role of TCF1 in reversing T cell exhaustion by CRISPR-HDR technology.